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found that the Aryl hydrocarbon receptor (AhR) on Th17 cells were activated upon CS exposure, and then resulted in the increased expression of EVs-loaded miR-132 derived from Th17 cells

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Populations assessed for uniqueness in this way were the three basal cell populations, KRT8 high , mature ciliated (with hybrid cells remove), SMG basal, SMG secretory, and surface secretory cell populations