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Description

TBC1D9B facilitates GTP hydrolysis by ARL8B Our collective results show that the TBC domain protein TBC1D9B directly binds to ARL8B in its GTP form, akin to the association of GAP proteins with their cognate active small GTPases

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However, the direct applicability of these findings to human risk remains uncertain, even though these findings raise concerns [44]

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J.LiR.et al (2018)

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IV glutathione removes such toxic compounds and aids the liver in performing its inherent detoxifying role

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