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Mechanisms in Depth: What GHK-Cu Does at the Cellular Level Even though the clinical case is unproven, the mechanistic biology is substantive and deserves a careful treatment, both because it explains the enthusiasm and because it defines what a future trial would be testing

We also examined the role of hepatic enzyme FMO3 on TMAO plasma levels

doi: 10.1093/clinchem/30.5.627 32

A promising therapeutic strategy is supplementation with vitamin E, based on a study showing that after supplementation with 350 mg vitamin E daily for 1 month, beta-thalassemia/hemoglobin patients with splenectomy (removal of the spleen) experienced a significant increase in their red blood cell membrane fluidity, suggesting that this approach could prevent red blood cell damage in this patient group (Sutipornpalangkul 2012)