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doi: 10.1186/s40478-025-01950-z 150

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The present study shows that exercise induces an increase in plasma CO levels, and this could mean that this defence mechanism has been activated by the increase in exercise-associated ROS and proinflammatory molecules, such as NO

Understanding the different types of 5-alpha reductase helps explain why localized inhibition matters

Concurrently, loss of Treg-mediated suppression permits Th17 activation and IL-17 release, driving robust neutrophil recruitment and destructive inflammation that promotes a more severe and refractory disease phenotype