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Its mechanism differs radically from BPC-157: GHK-Cu acts largely through chelation and redox modulation, delivering copper as a cofactor for lysyl oxidase and superoxide dismutase

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doi: 10.1210/clinem/dgaa863 Summary Keywords obesity, insulin-resistance, Tirzepatide, pathophysiology, GIP, GLP - 1, clinical trials, arterial hypertension Citation Corrao S, Pollicino C, Maggio D, Torres A and Argano C (2024) Tirzepatide against obesity and insulin-resistance: pathophysiological aspects and clinical evidence

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