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Common mechanisms include enhancing antioxidant defenses (typically through activation of the Nrf2/ARE pathway), inhibiting pro-inflammatory signaling (e.g., NF-B and MAPK pathways), increasing nitric oxide (NO) bioavailability in endothelial cells, regulating lipid metabolism, and alleviating pathological fibrosis

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Our results demonstrated that BPC-157 possessed wound healing effects on alkali-burned rats, and BPC-157 promotes proliferation, migration, and tube formation of human umbilical vein endothelial cells (HUVECs) through the extracellular signal-regulated kinases 1 and 2 (ERK1/2) signaling pathway

The SWISSPROT mouse proteome database of September 19 th , 2020, including validated splice variants was used (25,321 sequences), with sequences of common contaminants added

Neilipovitz, D