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As a synthetic agonist of proliferation-activated receptor (PPAR), TZDs exhibited no correlation between their anti-ferroptosis ability and PPAR--mediated gene transcription (Doll et al

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1) The results of the study performed by the research team of Park et al comprehensively detail the protective effects of GHK-Cu against LPS-induced inflammation and ALI, beginning with in vitro*experiments on RAW 264.7 macrophages and extending to an in vivo mouse model

In most infections, macrophages are the first to respond, sending signals to recruit other effector cells [16]

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