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Research shows it enhances fibroblast migration to injury sites through the FAK-paxillin pathway, improves collagen fiber organization and cross-linking, promotes angiogenesis to improve nutrient delivery, and reduces inflammatory mediators while preserving beneficial healing signals

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The mesenchymal phenotype of equine ASCs was confirmed by flow cytometry (FACS) using fluorochrome-conjugated monoclonal antibodies specific for CD44, CD45, CD90, and CD105 (Table 2) [36]