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Research demonstrates that these medicines affect early disease development by addressing critical pathophysiological mechanisms

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[1] [10] Additionally, Ozempic suppresses glucagon secretion, slows gastric emptying, and acts on appetite centres in the brain to promote satiety and reduce food intake

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Dysbiosis-induced increased intestinal permeability allows the translocation of bacterial endotoxins, notably LPS, into the systemic circulation, triggering inflammatory cascades that compromise thyroid tissue integrity and hormone synthesis