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By suppressing TGF-1 expression and preventing Smad2/3 phosphorylation, luteolin downregulates core ECM components, such as collagen I, fibronectin, and alpha-smooth muscle actin (-SMA), thereby limiting myofibroblast activation and pathological ECM accumulation

Enhanced mitochondrial biogenesis ameliorates disease phenotype in a full-length mouse model of Huntingtons disease

I see women abandon retinol in their fifties because their skin feels "too sensitive." Usually, the issue is not the retinol itself

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doi: 10.18203/2319-2003.ijbcp20150878 192