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Additionally, BPC-157 may modulate inflammatory responses by reducing pro-inflammatory mediators such as Nos2 and Nfkb while supporting anti-inflammatory processes

This highlights FGF21 and SLC25A39 as potential new intervention targets for mitigating mitochondrial damage and oxidative stress post-TBI, laying the groundwork for future clinical therapeutic strategies

Research on epithalon, particularly in older animal models, has shown improvements in immune cell function and antibody production

doi: 10.1016/j.micinf.2004.08.018 43 VerbeekRESiersemaPDVleggaarFPTen KateFJPosthumaGSouzaRFet al