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[DOI] [PubMed] [Google Scholar] 137.McKetin R., Dean O.M., Turner A., Kelly P.J., Quinn B., Lubman D.I., Dietze P., Carter G., Higgs P., Baker A.L., Sinclair B., Reid D., Manning V., Te Pas N., Liang W., Thomas T., Bathish R., Kent M., Raftery D., Arunogiri S., Cordaro F., Hill H., Berk M

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Indeed, if there is a methionine deficit, homocysteine can be re-methylated to form methionine, and if there is an adequate amount of methionine, homocysteine is used to produce cysteine (161)

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doi: 10.1016/j.prostaglandins.2009.04.001 63

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Mitochondrial & Metabolic Research Within mitochondrial and metabolic research settings, Glutathione is commonly investigated for its interaction with: Cellular-energy pathways Mitochondrial oxidative-stress regulation Cellular detoxification mechanisms Mitochondrial-function investigations Recovery-focused metabolic pathways These pathways have contributed to broader research interest in Glutathione within metabolic, mitochondrial-support, healthy-aging, and cellular-energy research models

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