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Pro-inflammatory cytokines activate chondrocytes to release catabolic factors, which impede the production of COL2, proteoglycans, and other synthetic metabolic components, hence facilitating chondrocyte catabolic processes and the degradation of articular cartilage [39,40]

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Part D plans participating in the model are required to place all model drugs on the same formulary tier, cover all medically accepted indications for model drugs, and apply the same cost sharing to all indications

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KD, by promoting the use of KBs as an energy source, offers considerable potential for neuroprotection in PD through mechanisms such as enhancing mitochondrial function, reducing oxidative stress, and exerting anti-inflammatory effects