The anti-endometriosis effects involve plentiful molecular mechanisms and targets, including anti-proliferative (CCNE1, CDK2, and CDK4), anti-inflammatory (PGE2, COX-2, TNF-, NO, IL-1, IL-6, and IL-8), anti-angiogenic (VEGF, matrix metalloproteinase and Ang-1), pro-apoptotic (Bax, Bak, Bcl-2, Bcl-xL), autophagy (Beclin 1, LC3-II/LC3-I), endoplasmic reticulum stress, endoplasmic reticulum apoptotic pathway, mitochondrial apoptotic pathway and caspase cascade, anti-migratory invasion (MMP-2, MMP-9, TIMP1, E-cadherin, N-cadherin, Snail and Slug), regulation of estrogen and progesterone receptors, and anti-fibrotic (E-calmodulin, -SMA, type I collagen, wave proteins and fibronectin)
Int J Mol Sci 23(10):15 Han FY, Conboy-Schmidt L, Rybachuk G, Volk HA, Zanghi B, Pan Y et al (2021) Dietary medium chain triglycerides for management of epilepsy: new data from human, dog, and rodent studies
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it is identical to the peptide core of CJC-1295 but lacks the Drug Affinity Complex (DAC) albumin-binding linker
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