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Cellular senescence is dynamic, often compromising normal tissue functionality by continuously secreting pro-inflammatory cytokines, chemokines, growth factors, and matrix metalloproteinasescollectively termed senescence-associated secretory phenotype (SASP) factors

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The amidated C-terminal residue differs across CT-family peptides and this residue forms the only direct contact with RAMPs in previously reported AMYR complexes 15,16

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