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An in-depth study of the mechanism suggested that it might, on the one hand, act on the PI3K/Akt/mTOR signaling pathway to downregulate the expression of VEGF, p-Akt, and p-mTOR, inhibit angiogenesis, and promote cell apoptosis (Xie et al., 2005), making it difficult to reach an effective therapeutic concentration

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1 H NMR (400 MHz, DMSO-d 6 ) = 5.85 (q, J = 6.3 Hz, 4H), 3.15 (hept, J = 6.9 Hz, 1H), 2.39 (s, 3H), 1.21 (d, J = 6.9 Hz, 6H)