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The deletion of these channels can reduce the nuclear factor B (NFB) phosphorylation (pro-apoptotic) and downregulate the expression of pro-apoptotic Bcl-2 family protein Bax (Chen et al., The activated microglia release Toll-like receptor 4, which is a pro-inflammatory protein activating the NFB pathway

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The rate of reaction depends on the cellular GSH level and the ratio of GSH to H 2 O 2 concentrations [100]

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