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This creates feed-forward cycles that exacerbate cuproptosis: mtROS oxidize cytosolic GSH, releasing bound Cu, and may upregulate the copper importer CTR1 via pathways such as NF-B, increasing copper influx [65]

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That is where phase III comes in moving the phase II conjugated metabolites across cell membranes and out toward the bile or urine

Treatment of copper poisoning is mainly symptomatic, and besides that detoxification procedures like chelation therapy, hemodialysis, and plasma exchanges might be necessary based on the case