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AGEs may also promote vascular dysfunction by reducing the bioavailability of nitric oxide (NO) and inhibiting endothelial NO synthase and antioxidant defense mechanisms 13,14

Neuroimaging studies have consistently demonstrated reduced D2R availability, reflected by decreased binding potential, in key striatal and extrastriatal regions of obese individuals, supporting an association between obesity, central dopaminergic hypofunction, and dysregulated prolactin homeostasis ( 5 Mechanisms by which obesity may promote hyperprolactinemia We have systematically analyzed the potential mechanisms by which hyperprolactinemia promotes obesity

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Certain meats and seafood are higher in uric acid