Metabolism of aflatoxins: key enzymes and interindividual as well as interspecies differences
Consensus criteria for diagnosis, staging, and treatment response assessment of T-cell prolymphocytic leukemia
High-Quality Manufacturing: Produced in an FSSAI-approved, ISO 22000:2018 certified, WHO-GMP & GMP-compliant facility under stringent quality standards
Paracetamol Paracetamol-induced hepatotoxicity represents the result of an imbalance between metabolic detoxication of paracetamol, via conjugation with glucuronic acid and sulphate, and metabolic activation to an unstable toxic metabolite ( N -acetyl- p -benzoquinone imine, NAPQI) via oxidation by cytochrome P450
9,13 GSH repletion not only provides surplus cysteine as an energy substrate for the Krebs cycle, it also serves the important role of scavenging for free radicals and peroxynitrite