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doi: Pongrojpaw, D., Somprasit, C
The experiment demonstrates the potential of IGF-I treatment of catabolic states and especially the value of modified forms of growth factors that bind weakly to IGF-binding proteins. In another study in the Journal of Developmental Cell , IGF-1 LR3 was shown to help prevent muscle wasting and stimulate muscle growth through the activation of the IGF1R/IRS1/PI3K/Akt pathway
Trans-membrane tight junction proteins are linked to the actin-based cytoskeleton via zonula occludens-1, zonula occludens-2, zonula occludens-3, cingulin, acute lymphoblastic leukemia-1 fusion partner in chromosome 6, and 7H6 (also named barmotin), which may also play an essential role in modulating tight junction tightness [11]
( Supplementary Figure S1-31
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