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These studies underscore the paradoxical role of arginine metabolism and availability depending on the circumstances (e.g., immune cells infiltration and the type of arginine metabolizing enzymes expressed in these cells in the TME) and type of tumor, implying that arginine supplementation might be interesting for some, but not other tumors (Table 3)

Although emerging evidence suggests that both aging and chronic hyperglycemia activate the HBP in ECs, the direct impact of age-associated HBP remodeling on vascular dysfunction remains insufficiently characterized

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Identifying these differences through advanced molecular and imaging techniques could help tailor ferroptosis-targeted interventions to specific subphenotypes, optimizing therapeutic efficacy

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