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This study suggests that the mechanism driving soluble UA-induced inflammation is dependent on redox state changes along with mitochondrial oxidant production [Citation20]

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The cellular concentration of GSH, therefore, is regulated by a complex process of precursor amino acid transport across cell membranes, intracellular synthesizing enzymes, feedback regulation, and intracellular GSH complexing via conjugation of GSH with a variety of electrophilic compounds through GSH transferase reactions