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Future mitochondria-directed antioxidants for AP will need to avoid strong membrane-perturbing cations, be optimized specifically for acinar-cell bioenergetics, and be evaluated not only for redox effects but also for their impact on mitochondrial ATP production and Ca 2+ handling

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Concomitantly, clinical and experimental observations 70,71 have revealed that endogenous estrogens can protect against IR primarily through ER- activation in multiple tissues, including in the brain, liver, skeletal muscle, and adipose tissue, in addition to pancreatic cells