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The apoptosis rate was significantly increased, as well as the release of inflammatory factorsIL-1 and tumor necrosis factor (TNF)- [112]

This sophisticated crosstalk, where parallel pro-apoptotic pathways (ATF4-driven induction and CHOP-mediated fine-tuning) converge on the CHAC1 promoter, allows for precise control of cell fate under prolonged ER stress (Nomura et al., 2020)

A., Tibar, H., Regragui, W

Defects in CI are among the most common causes of mitochondrial disorders and have been linked to a wide range of pathologies, including neurodegenerative and cardiac diseases 28

Cytokine modulation by IL-35 in mice with allergic rhinitis