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However, liver enlargement and regeneration activated by PXR cause YAP to undergo deacetylation and K63-linked ubiquitination in a Sirt2-dependent manner, which allows YAP-TEAD binding in the nucleus

doi: 10.1094/MPMI-03-22-0063-R 13 Diaz-VivancosP.WolffT.MarkovicJ.PallardF

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We then hypothesized that the inefficacy of the peptide in inducing cell death in both types of cells might be attributed to inadequate cellular uptake