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found that epithalon elongated telomeres and induced telomerase activity when introduced to telomerase-negative human fetal fibroblast cultures, with the authors concluding that epithalon may prolong the lifespan of cell populations [2]
Disruption of mitochondrial function is a key factor in Parkinsons disease (PD) pathogenesis
This is characterized by increased expression of pro-inflammatory genes (IL-1, IL-6 and TNF-) and decreased expression of anti-viral and antibody genes (e.g., IFNs) [71]
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