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Several studies have shown that METTL3 plays important regulatory roles in prostate cancer proliferation, migration, invasion, apoptosis, drug resistance androgen-induced splicing and glycolipid metabolism maintenance (125,126)

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While these compounds differ in their molecular structure, they share common mechanisms of action in combating oxidative stress, which are particularly relevant to ED where oxidative stress significantly impairs endothelial cell function and NO signaling [8]

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