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Most published data derive from small pilot studies, in vitro experiments, and animal research, which substantially limits the conclusions that can be drawn

However, an alternative explanation, albeit not mutually exclusive, is direct interaction of GLP-1RAs with GLP-1R in brain regions such as the brainstem (area postrema, nucleus tractus solitarii), which are not protected by the blood-brain barrier, and are typically involved in medication-induced nausea (chemosensitive area) (5456)

For GLP-1 users, the aim is quality over quantity choosing foods that sustain fullness, help regulate blood glucose levels, and provide concentrated nutrition (4)