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The best understood effectors for this process are PDE3B and ABHD15 that operated by the suppression of cAMP to attenuate pro-lipolytic PKA signaling toward adipose triglyceride lipase (ATGL), hormone-sensitive lipase (HSL), and perilipin (PLIN)

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Research Applications Compared Because the mechanisms differ, the two compounds have accumulated their strongest evidence in somewhat different areas with meaningful overlap in wound healing and angiogenesis