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More specifically, it has been reported that following ischemia, oxidative stress, or acidosis injury in rat caECs, CMs, and aortic SMCs, sAC-dependent apoptosis is triggered by translocation of sAC from the cytosol to mitochondria with submitochondrial PKA activation, Bax phosphorylation, and cytochrome c release (4548)

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[DOI] [PMC free article] [PubMed] [Google Scholar] Romagnani P, Remuzzi G, Glassock R, Levin A, Jager KJ, Tonelli M, Massy Z, Wanner C, Anders HJ

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