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Abstract Background Transthyretin (TTR) amyloidosis is a progressive, life-threatening disorder caused by extracellular deposition of amyloid fibrils derived from the plasma protein TTR

Some researchers believe mitochondrial dysfunction in children could be one of the many contributing factors , while others think it may be a result of other imbalances

Exacerbation of sensorimotor dysfunction in mice deficient in Atp13a2 and overexpressing human wildtype alpha-synuclein

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