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Scalise V, Balia C, Cianchetti S, et al
Multiple preclinical studies have demonstrated that co-targeting glycolysis and mitochondrial metabolism, or modulating key metabolic regulators such as AMPK, can significantly enhance the sensitivity of CRC cells to chemotherapy and targeted agents
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M., Jabra-Rizk, M
Thus, dysbiosis drives kidney damage through toxin release, barrier failure, and inflammation, while gut microecological preparations weaken the key mechanisms that regulate renal inflammation and fibrosis
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