Initially, it looked to me like homocysteine might be an important storage/transfer molecule and the factors that cause its elevation are the actual problem, not the homocysteine itself
It is highly bioavailable, rapidly absorbed through the intestinal wall, and reaches peak concentrations in the blood plasma and cerebrospinal fluid within roughly 2 to 4 hours post-ingestion
Unlike IGF-1 DES, which benefits from site-specific injection, LR3's long half-life means injection site matters less, the peptide will circulate systemically regardless of where you inject it
[DOI] [PubMed] [Google Scholar] 101.Van Nieuwpoort I.C., Vlot M.C., Schaap L.A., Lips P., Drent M.L
Increased brain concentrations of a neuroinhibitory steroid in human hepatic encephalopathy