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Minoxidil and GHK-Cu share VEGF upregulation but through different pathways (minoxidil via potassium channel/PHD inhibition, GHK-Cu via fibroblast stimulation)

They concluded that topical treatment at the defect site appeared to stimulate burn healing more effectively than injection [22]

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The post-synaptic currents, which were recorded in the presence of strychnine, picrotoxin and tetrodotoxin, represented spontaneous bursts likely mediated by AMPA receptors