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Similar to research on LDL-C levels and how the use of GLP-1 RAs such as liraglutide and exenatide can affect it, reports found that liraglutide and exenatide treatment also has insignificant effects on HDL-C [66-68]

At 30-month follow-up, despite weight regain in all groups (WL + AT: 7.1 kg

Because ipamorelin and GHRH analogs act through separate receptor pathways (GHS-R1a versus the GHRH receptor), preclinical research has investigated the two compound classes together, hypothesizing that dual-pathway stimulation may produce additive or synergistic effects on GH release compared with either pathway alone a design that leverages the cAMP/calcium signaling distinction described above

Considering the broader genetic background, pharmacogenomic variability may further contribute to treatment heterogeneity, as genetic variants affecting GLP-1 receptor signaling, appetite regulation, insulin secretion, and hostmicrobiota interactions could influence individual responsiveness (126)

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