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Research highlights the crucial role of oxidative stress in the pathophysiology of PD [50, 66]

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Specifically, IgGs obtained from the CSF of patients with anti-GAD65Ab-associated CA depressed GABA release in cerebellar brain slices [77, 78, 82, 85], and their intracerebellar injection interfered with cerebellar control of the motor cortex and resulted in ataxic gait in rats [79]

Repeated-dose oral N-Acetylcysteine in Parkinsons disease: Pharmacokinetics and effect on brain glutathione and oxidative stress

This decline can contribute to fatigue, muscle loss, poor sleep, and stubborn fat accumulation