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cytosolic calcium instability drives mitochondrial calcium overload, destabilizes membrane potential, increases permeability transition pore opening, and amplifies ROS generation

The C-terminal difference between the two variants makes GAC more specifically active, which may contribute to its greater susceptibility to overexpression in aggressive cancers (Redis et al., 2016)

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Renal blood perfusion decreased significantly by 6 h and reached its lowest point at 24 h, with glycocalyx thinning becoming evident [21]