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The persistent challenge with sermorelin is enzymatic stability: dipeptidyl peptidase-IV (DPP-IV) cleaves the Tyr1-Ala2 bond within minutes of administration, producing the inactive GHRH(3-29) fragment and limiting the practical research applications of the unmodified peptide

2A) [36]

Impairment in Glutathione peroxidase 4 (GPX4) function, which attenuates cellular redox defense, i s also associated with cartilage extracellular matrix (ECM) damage (Miao et al., 2022) implying targeting ferroptosis as a promising new direction for possible interventions in OA pathogenesis