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Although the use of senolytics alone are being investigated as anti-ageing approaches (121), their potential off-target effects and how these therapies may affect physiological wound healing have yet to be elucidated

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indeed, with respect to malaria protection, it has been theorized that protection conferred by G6PD deficiency may be related to increased phagocytosis of parasitized oxidatively-damaged G6PD-deficient RBCs (7779) via binding by macrophage Fc and lectin-like receptors (80), and that such parasites may be more susceptible to ROS (81)