The disruption of this microbiota-SCFAs-host axis not only affects SCFAs metabolism in the enterohepatic circulation but also exacerbates hepatic insulin resistance and lipid deposition through gut-liver axis immune crosstalk, forming a vicious cycle that drives progression from non-alcoholic steatohepatitis (NASH) to cirrhosis [36]
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However, the incidence of gastrointestinal events, discontinuations due to adverse events, and mean increase in pulse rate were higher with orforglipron than oral semaglutide
Treatment of animals Four groups of Sprague-Dawley rats housed under the same conditions and fed on the same diet were used in this experiment
Key highlights Both semaglutide and dulaglutide are GLP-1 medications with the same mechanism of action
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