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Aging impairs IGF-I receptor activation and induces skeletal resistance to IGF-I

(ii) can be converted to neutral TG, packaged into very-low-density lipoprotein cholesterol (VLDL-C) particles and secreted into the circulation, while intrahepatic TG are stored as intracellular lipid droplets contributing to the fatty acid pool inside hepatocytes and (iii) glycerol substrates and odd chain NEFAs can be used during gluconeogenesis