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In white adipose tissue of obese, insulin-resistant and aged mammals, NNMT is dramatically upregulated and because it consumes both the universal methyl donor (SAM) and the NAD precursor (nicotinamide), its hyperactivity simultaneously depletes the cellular methylation reserve and the NAD-salvage pool [1,2]

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Your body also needs energy to function and be productive when youre awake

Better bone strength: It aids calcium absorption, reducing the risk of bone loss and supporting skeletal health

Limited long-term data: most evidence is from animal models, with no long-term human study of NNMT inhibition and oncological endpoints