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The polymorphism is caused by a single point mutation (G to A nucleotide substitution), leading to a substitution of lysine for glutamate at position 487 (E487K) in the ALDH2 monomer [5]

With its extended half-life growth factor analog properties, it provides prolonged activity, ensuring sustained effects on muscle growth and metabolic efficiency

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Efficacy of empagliflozin as adjunctive therapy to citalopram in major depressive disorder: a randomized double-blind, placebo-controlled clinical trial