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Mitochondrial dysfunction in huntingtons disease: Pathogenesis and therapeutic opportunities

Under conditions of dysbiosis, it may contribute to bone loss in OP by modifying immune function and increasing the production of bone-metabolism-related cytokines

While further research is needed to clarify the causal pathways, the potential role of autoimmunity as a driver of persistent symptoms should not be overlooked in clinical practice

Colchicine also has a major role when patients begin therapy with allopurinol (see below) to prevent the increase in gout attacks that can happen when allopurinol is begun

Exploring the role of parthanatos in CNS injury: molecular insights and therapeutic approaches