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Documented antitumor mechanisms include suppression of excessive inflammation and potential functional restoration of CD8+ T cells, reduction of NET formation in neutrophils to enhance CD8+ T cell cytotoxicity, direct restoration of NK cell function with indirect promotion of N1-polarized neutrophils, and promotion of DC maturation and antigen presentation to reinforce T-cell activation
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The activities of nnAbs against molecules expressed on the surface of infected cells but not on virions, such as the NS1 protein of dengue virus, are readily understood
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