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Gastrointestinal and Enteric Research As a derivative of a gastric-origin protein, BPC-157 has been studied across gastric ulcer, intestinal permeability, NSAID-induced GI damage, and colitis preclinical models

FAK-paxillin pathway: Cell culture work suggests BPC-157 activates focal adhesion kinase (FAK) and paxillin, proteins involved in cell migration important for wound closure

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