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Collectively, these findings underscore both the therapeutic potential and the uncertainties associated with translating single-agent antifibrotic therapies into routine clinical practice
(6) Lango R, et al
According to Pande et al., dietary thyroxine could cause an increase in carnitine levels in the liver and serum, while carnitine levels in the heart, skeletal muscle, and the urinary excretion of carnitine remained unaffected in experimental rats [27]
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