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Lipid-induced TLR-4 expression and subsequent secretion of tumor necrosis factor- activate the NF-kb signaling pathway, resulting in the release of pro-inflammatory factors and promoting the proliferation and steroidogenic activities, which in turn stimulate excess ovarian androgen production

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T4 is the most abundant member of the beta-thymosin family in mammalian tissue and the principal G-actin sequestering molecule in eukaryotic cells, present in blood platelets, wound fluid and essentially all nucleated cells