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Altogether, we conclude that HSV-1 infection can enhance Keap1-dependent Nrf2 degradation, thereby disrupting cellular redox homeostasis and promoting ferroptosis
Since HEDS contains no glutathione moiety, alterations at the scaffold site could provide further insights regarding GSSEtOH as a potential reaction intermediate
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Red Blood Cell Formation: It aids in the creation of red blood cells, reducing the risk of anemia
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